When APOM is low: how it can harm kidneys in Alport and related glomerular diseases

APOM deficiency contributes to renal failure in glomerular diseases

NIH-funded research University of Miami School of Medicine · NIH-11361041

This project looks at whether low levels of the protein APOM cause lipid buildup that damages podocytes and speeds kidney failure in people with Alport syndrome and other glomerular diseases.

Quick facts

Grant typeR01 grant
Study typeNIH-funded research
Funding institutionUniversity of Miami School of Medicine NIH-funded
Lab location1 site (Coral Gables, United States)
Project IDNIH-11361041 on NIH RePORTER

What this research studies

From a patient point of view, the team will compare APOM levels and lipid handling in kidney tissue and blood from people with Alport and other glomerular diseases to patterns seen in animal and cell models. They will study gene expression in patient samples and in Col4a3 knockout mice and cultured podocytes to see how APOM, sphingosine-1-phosphate (S1P), and cholesterol interact. The researchers will test whether restoring APOM function or targeting S1P-related pathways reduces cholesterol and sphingolipid buildup and protects podocytes in models. Findings from human samples and experiments will be combined to identify druggable targets that could be tested in future therapies.

Who could benefit from this research

Good fit: Ideal candidates would be people with Alport syndrome or other glomerular kidney diseases who are followed at participating centers or who contribute samples to research cohorts.

Not a fit: People without glomerular disease or whose kidney problems arise from unrelated causes are unlikely to benefit directly from this research.

Why it matters

Potential benefit: If successful, this work could point to new treatments that protect podocytes and slow kidney failure in Alport syndrome and other glomerular diseases.

How similar studies have performed: Previous research has linked disrupted cholesterol transport and sphingolipid metabolism to podocyte injury, but targeting APOM/S1P in human kidney disease remains a new and unproven approach.

Where this research is happening

Coral Gables, United States

Researchers

About this research

  1. This is an active NIH-funded research project — typically early-stage science, not a clinical trial accepting patient enrollment.
  2. Some NIH-funded labs run parallel clinical studies or seek volunteers for related work. To check, contact the principal investigator or institution listed above.
  3. For full project details, budget, and progress reports, visit the official NIH RePORTER page below.
Conditions Alport syndromeAlport syndrome (AS, ATS)Alport syndrome-like hereditary nephritisAlport syndrome-like hereditary nephritis (ASLHN, ASLN)Alport's Syndrome
Last reviewed 2026-06-13 by the Find a Trial editorial team. Information on this page is for educational purposes and is not medical advice. Always consult qualified healthcare professionals about clinical trial participation.