New drugs that target the GABAA α5 receptor to improve memory and thinking in schizophrenia

A preclinical therapeutic platform to develop GABAA alpha5 receptor positive allosteric modulators to improve cognitive function in schizophrenia

NIH-funded research Johns Hopkins University · NIH-11145807

Developing compounds that boost a specific brain receptor (GABAA α5) to help people with schizophrenia improve memory and other thinking skills.

Quick facts

Grant typeR21 grant
Study typeNIH-funded research
Funding institutionJohns Hopkins University NIH-funded
Lab location1 site (Baltimore, United States)
Project IDNIH-11145807 on NIH RePORTER

What this research studies

This project works in the lab to create and test drug-like molecules that positively modulate the GABAA α5 receptor, a protein common in the hippocampus that helps control brain excitation. Researchers will use cell-based tests and animal models to measure how selectively these compounds act on the α5 subunit and whether they restore the balance between excitation and inhibition in hippocampal circuits. The team will evaluate potency, selectivity, and effects on memory-related behaviors in preclinical models and optimize lead candidates for possible future clinical testing. At this stage the work is preclinical and does not involve giving these drugs to people, but it focuses on a mechanism tied to cognitive problems in schizophrenia.

Who could benefit from this research

Good fit: People with schizophrenia who have noticeable memory or thinking difficulties, particularly those thought to involve hippocampal overactivity, would be the most likely candidates for future trials stemming from this work.

Not a fit: People whose cognitive problems are caused primarily by medication side effects, other psychiatric or neurological disorders, or unrelated medical conditions may not benefit from this approach.

Why it matters

Potential benefit: If successful, these drugs could improve memory and other cognitive abilities that current schizophrenia treatments do not effectively treat.

How similar studies have performed: Preclinical animal and cellular studies of GABAA α5 modulation have shown promise for improving memory by restoring inhibitory signaling, but human clinical evidence remains limited.

Where this research is happening

Baltimore, United States

Researchers

About this research

  1. This is an active NIH-funded research project — typically early-stage science, not a clinical trial accepting patient enrollment.
  2. Some NIH-funded labs run parallel clinical studies or seek volunteers for related work. To check, contact the principal investigator or institution listed above.
  3. For full project details, budget, and progress reports, visit the official NIH RePORTER page below.
Conditions CNS DiseasesCNS disorderCentral Nervous System DiseasesCentral Nervous System Disorders
Last reviewed 2026-06-13 by the Find a Trial editorial team. Information on this page is for educational purposes and is not medical advice. Always consult qualified healthcare professionals about clinical trial participation.