Stopping genetic changes that cause colorectal cancer to stop responding to targeted drugs

Targeting Chromosomal Instability in the Evolution of Resistance to Matched Therapies Against Colorectal Cancer to Extend Treatment Response

NIH-funded research Sloan-Kettering Inst Can Research · NIH-11291328

Tests ways to stop genetic changes in colorectal cancer that make targeted drugs stop working for people with advanced disease.

Quick facts

Grant typeR21 grant
Study typeNIH-funded research
Funding institutionSloan-Kettering Inst Can Research NIH-funded
Lab location1 site (New York, United States)
Project IDNIH-11291328 on NIH RePORTER

What this research studies

As a patient, I would learn whether my tumor has extra copies of cancer genes (sometimes carried on mobile pieces of DNA called ecDNA) that make targeted drugs stop working. The researchers will study tumor samples from patients and laboratory models to see when these amplifications appear and how they change with treatment. They plan to identify weaknesses created by chromosomal instability that could be targeted with additional therapies to keep drugs working longer. Finding markers of early resistance could help guide better treatment choices for patients like me.

Who could benefit from this research

Good fit: People with metastatic colorectal cancer who are receiving or about to receive targeted therapies (for example BRAF- or KRAS G12C-directed drugs), especially if their tumor or blood tests show gene amplifications, would be the best match.

Not a fit: Patients with early-stage colorectal cancer treated only with surgery or those without targetable driver mutations are unlikely to benefit directly from this research.

Why it matters

Potential benefit: If successful, this work could help targeted treatments keep working longer and delay cancer progression for patients with metastatic colorectal cancer.

How similar studies have performed: Prior studies have shown gene amplifications and ecDNA can drive rapid resistance in colorectal cancer, but using chromosomal instability as a treatable vulnerability is a newer, experimental idea.

Where this research is happening

New York, United States

Researchers

About this research

  1. This is an active NIH-funded research project — typically early-stage science, not a clinical trial accepting patient enrollment.
  2. Some NIH-funded labs run parallel clinical studies or seek volunteers for related work. To check, contact the principal investigator or institution listed above.
  3. For full project details, budget, and progress reports, visit the official NIH RePORTER page below.
Conditions Cancer CauseCancer EtiologyCancer GenesCancer-Promoting GeneCancers
Last reviewed 2026-06-13 by the Find a Trial editorial team. Information on this page is for educational purposes and is not medical advice. Always consult qualified healthcare professionals about clinical trial participation.