Stopping abnormal blood vessel growth and scar tissue under the retina in wet age-related macular degeneration

Inhibiting Neovascularization and Subretinal Fibrosis in Neovascular Age-Related Macular Degeneration

NIH-funded research Utah State Higher Education System--University of Utah · NIH-11142546

This project tests new drug approaches to prevent abnormal blood vessels and scarring under the retina in adults with neovascular (wet) age-related macular degeneration.

Quick facts

Grant typeR01 grant
Study typeNIH-funded research
Funding institutionUtah State Higher Education System--University of Utah NIH-funded
Lab location1 site (Salt Lake City, United States)
Project IDNIH-11142546 on NIH RePORTER

What this research studies

From a patient's point of view, researchers are studying why scar tissue forms under the retina in wet AMD by looking at how retinal and blood-vessel cells change their behavior (EMT and EndoMT). They will test molecules that may block or reverse those cell changes in lab models and preclinical systems, aiming to keep the endothelial barrier intact. The work is intended to complement existing anti-VEGF treatments and to work when anti-VEGF alone stops controlling disease progression. Successful approaches would then move toward drug development and possible clinical testing.

Who could benefit from this research

Good fit: Adults (21+) with neovascular (wet) age-related macular degeneration, especially those showing early subretinal fibrosis or reduced response to anti-VEGF treatments, would be the most relevant candidates.

Not a fit: People with non-neovascular (dry) AMD or those with long-standing, end-stage scarring and irreversible vision loss are unlikely to benefit from these approaches.

Why it matters

Potential benefit: If successful, this work could reduce or prevent subretinal scarring and help preserve vision in people with neovascular AMD beyond current anti-VEGF therapy.

How similar studies have performed: Anti-VEGF treatments reliably shrink abnormal vessels but do not stop fibrosis, and targeting EMT/EndoMT is a newer approach with promising preclinical results but limited clinical proof so far.

Where this research is happening

Salt Lake City, United States

Researchers

About this research

  1. This is an active NIH-funded research project — typically early-stage science, not a clinical trial accepting patient enrollment.
  2. Some NIH-funded labs run parallel clinical studies or seek volunteers for related work. To check, contact the principal investigator or institution listed above.
  3. For full project details, budget, and progress reports, visit the official NIH RePORTER page below.
Last reviewed 2026-06-10 by the Find a Trial editorial team. Information on this page is for educational purposes and is not medical advice. Always consult qualified healthcare professionals about clinical trial participation.