Metabolism of immune-suppressing cells in pancreatic tumors

Metabolic regulation of immunosuppressive myeloid cells in the tumor microenvironment of pancreatic cancer.

NIH-funded research University of Michigan at Ann Arbor · NIH-11317119

This work looks at how certain immune cells in pancreatic cancer use metabolic signals to shut down anti-cancer T cells, with the goal of finding ways to help patients' immune systems fight their tumors.

Quick facts

Grant typeR01 grant
Study typeNIH-funded research
Funding institutionUniversity of Michigan at Ann Arbor NIH-funded
Lab location1 site (Ann Arbor, United States)
Project IDNIH-11317119 on NIH RePORTER

What this research studies

Researchers will use mouse models of pancreatic ductal adenocarcinoma to study immune-suppressing myeloid cells that gather in tumors. They will focus on the CCR1 receptor, the myeloid enzyme IRG1, and the metabolite itaconate to see how these factors change tumor growth and CD8+ T cell activity. The team will remove or block CCR1 or IRG1 in myeloid cells in orthotopic mouse tumors and measure effects on tumor size, immune cell infiltration, and metabolite levels. Findings are intended to point toward drug targets that could reduce immunosuppression in human pancreatic tumors and make immunotherapies work better.

Who could benefit from this research

Good fit: People with pancreatic ductal adenocarcinoma, particularly those whose tumors are resistant to current immunotherapy approaches, would be the most relevant patient group for future related trials.

Not a fit: Patients without pancreatic cancer or whose tumors do not rely on myeloid-driven immunosuppression are unlikely to benefit from these specific findings.

Why it matters

Potential benefit: If successful, this research could identify new targets to lower immune suppression in pancreatic tumors and improve the effectiveness of immunotherapy for patients.

How similar studies have performed: Early preclinical mouse studies removing CCR1 or IRG1 have reduced tumor growth and increased CD8+ T cell infiltration, but translating these results to effective human treatments remains unproven.

Where this research is happening

Ann Arbor, United States

Researchers

About this research

  1. This is an active NIH-funded research project — typically early-stage science, not a clinical trial accepting patient enrollment.
  2. Some NIH-funded labs run parallel clinical studies or seek volunteers for related work. To check, contact the principal investigator or institution listed above.
  3. For full project details, budget, and progress reports, visit the official NIH RePORTER page below.
Conditions Animal Cancer Model
Last reviewed 2026-06-10 by the Find a Trial editorial team. Information on this page is for educational purposes and is not medical advice. Always consult qualified healthcare professionals about clinical trial participation.