How Trichomonas vaginalis harms protective vaginal bacteria and triggers inflammation

RP-Riestra/Sussman: Investigating the Antibacterial and Immune Modulating Effects of Trichomonas Vaginalis Infection and Pyroptosis

NIH-funded research San Diego State University · NIH-11313847

This work looks at whether the sexually transmitted parasite Trichomonas vaginalis kills helpful lactobacilli and causes inflammatory cell death in people with vaginal infections.

Quick facts

Grant typeNIH-funded research
Study typeNIH-funded research
Funding institutionSan Diego State University NIH-funded
Lab location1 site (San Diego, United States)
Project IDNIH-11313847 on NIH RePORTER

What this research studies

Scientists will watch live interactions between the parasite and protective vaginal bacteria and measure bacterial death with staining methods. They will block parasite processes and make parasites missing a key protein to test whether the parasite engulfs or digests lactobacilli. The team will also test a fragment of the human protein gasdermin D to see if it can kill lactobacilli and use cells without gasdermin D to see how inflammatory cell death affects bacteria. Together these lab experiments aim to explain how infection can lead to bacterial imbalance and inflammation in the female reproductive tract.

Who could benefit from this research

Good fit: People with current or recurrent trichomoniasis or bacterial vaginosis, or those willing to provide cervicovaginal samples, would be the most relevant participants for related efforts.

Not a fit: People without vaginal infections or those unable to provide samples are unlikely to gain direct benefits from this primarily laboratory-focused research.

Why it matters

Potential benefit: If successful, this work could reveal mechanisms that lead to bacterial vaginosis during trichomoniasis and point to new prevention or treatment strategies to protect vaginal health.

How similar studies have performed: Previous studies have shown T. vaginalis can disrupt lactobacilli and cause inflammation, but using pyroptosis and gasdermin D as an antibacterial mechanism is a newer approach with mainly preclinical evidence.

Where this research is happening

San Diego, United States

Researchers

About this research

  1. This is an active NIH-funded research project — typically early-stage science, not a clinical trial accepting patient enrollment.
  2. Some NIH-funded labs run parallel clinical studies or seek volunteers for related work. To check, contact the principal investigator or institution listed above.
  3. For full project details, budget, and progress reports, visit the official NIH RePORTER page below.
Last reviewed 2026-06-13 by the Find a Trial editorial team. Information on this page is for educational purposes and is not medical advice. Always consult qualified healthcare professionals about clinical trial participation.