How pancreatic cancer cells scavenge nutrients when starving

Regulation of Nutrient Stress-Induced Macropinocytosis in Pancreatic Ductal Adenocarcinoma

NIH-funded research Sanford Burnham Prebys Medical Discovery Institute · NIH-11145627

This work looks at how pancreatic cancer cells increase protein uptake to survive low-nutrient conditions, aiming to help people with pancreatic cancer.

Quick facts

Grant typeR01 grant
Study typeNIH-funded research
Funding institutionSanford Burnham Prebys Medical Discovery Institute NIH-funded
Lab location1 site (La Jolla, United States)
Project IDNIH-11145627 on NIH RePORTER

What this research studies

From a patient perspective, the team is studying how pancreatic ductal adenocarcinoma cells use a process called macropinocytosis to swallow and break down proteins as a way to get amino acids when food is scarce. They compare what happens when tumors run low on glutamine and when cells are exposed to a glutamine-like drug, and they map which signaling proteins turn the process on. Researchers use lab-grown cancer cells and animal models to trace the pathways and test how blocking these routes affects tumor growth. The goal is to find targets that could stop tumors from 'feeding' themselves and make other treatments work better.

Who could benefit from this research

Good fit: People with pancreatic ductal adenocarcinoma, particularly those whose tumors rely on glutamine metabolism, would be the most relevant group for future trials based on this research.

Not a fit: Patients with cancers other than pancreatic ductal adenocarcinoma or whose tumors do not use macropinocytosis for nutrient uptake are less likely to benefit from findings here.

Why it matters

Potential benefit: If successful, this work could point to new ways to block tumor nutrient uptake and weaken pancreatic tumors so treatments control growth more effectively.

How similar studies have performed: Prior research has shown pancreatic cancers use macropinocytosis and that targeting glutamine metabolism can affect tumor growth, but some signaling links explored here are relatively new.

Where this research is happening

La Jolla, United States

Researchers

About this research

  1. This is an active NIH-funded research project — typically early-stage science, not a clinical trial accepting patient enrollment.
  2. Some NIH-funded labs run parallel clinical studies or seek volunteers for related work. To check, contact the principal investigator or institution listed above.
  3. For full project details, budget, and progress reports, visit the official NIH RePORTER page below.
Conditions Cancer CauseCancer EtiologyCancer GenesCancer TreatmentCancer-Promoting Gene
Last reviewed 2026-06-13 by the Find a Trial editorial team. Information on this page is for educational purposes and is not medical advice. Always consult qualified healthcare professionals about clinical trial participation.