How mutant p53 helps cancers hide from the immune system

Oncogenic functions of mutant p53

NIH-funded research State University New York Stony Brook · NIH-11247954

Researchers are looking at how specific changes in the p53 gene let cancer cells block an internal immune alarm system, with the goal of helping people whose tumors carry p53 mutations.

Quick facts

Grant typeR01 grant
Study typeNIH-funded research
Funding institutionState University New York Stony Brook NIH-funded
Lab location1 site (Stony Brook, United States)
Project IDNIH-11247954 on NIH RePORTER

What this research studies

If I have cancer with a p53 mutation, this work tries to explain how that altered p53 protein stops the cGAS/STING immune signaling that would normally flag damaged cells. The team uses laboratory experiments to study how mutant p53 changes signaling inside cancer cells and how those changes affect immune responses around the tumor. They will map the molecular steps involved and study the consequences for tumor behavior. The results are intended to guide future treatments that consider a tumor's p53 status.

Who could benefit from this research

Good fit: People whose tumors are known to carry p53 missense mutations (or who have cancers with high chromosomal instability) would be the most relevant group for findings from this work and potential future trials.

Not a fit: Patients with cancers that retain normal (wild-type) p53 or whose disease is driven by unrelated mechanisms are less likely to benefit directly from this specific line of research.

Why it matters

Potential benefit: If successful, this could point to new ways to restore immune detection of tumors or to choose therapies based on whether a patient's tumor has mutant p53.

How similar studies have performed: Prior studies show mutant p53 promotes cancer growth and cGAS/STING activates anti-tumor immunity, but connecting mutant p53 to suppression of cGAS/STING is a relatively new and developing area.

Where this research is happening

Stony Brook, United States

Researchers

About this research

  1. This is an active NIH-funded research project — typically early-stage science, not a clinical trial accepting patient enrollment.
  2. Some NIH-funded labs run parallel clinical studies or seek volunteers for related work. To check, contact the principal investigator or institution listed above.
  3. For full project details, budget, and progress reports, visit the official NIH RePORTER page below.
Conditions Cancer GenesCancer Suppressor GenesCancer-Promoting GeneCancers
Last reviewed 2026-06-13 by the Find a Trial editorial team. Information on this page is for educational purposes and is not medical advice. Always consult qualified healthcare professionals about clinical trial participation.