How kidneys use branched-chain amino acids for energy in tubular cells

Role of branched-chain amino acid catabolism in the proximal tubule

NIH-funded research State University New York Stony Brook · NIH-11319848

Researchers aim to boost how proximal tubule cells in the kidney break down branched‑chain amino acids to help protect people from acute kidney injury and its progression to chronic kidney disease.

Quick facts

Grant typeR01 grant
Study typeNIH-funded research
Funding institutionState University New York Stony Brook NIH-funded
Lab location1 site (Stony Brook, United States)
Project IDNIH-11319848 on NIH RePORTER

What this research studies

This project looks at the proximal tubule — the kidney cells most affected during acute kidney injury — and how they use branched‑chain amino acids (BCAAs) to feed energy-producing pathways. The team will combine cell and animal experiments with analysis of human kidney data and tissue to see whether reduced BCAA breakdown causes loss of cellular ATP and worse injury. They will test interventions that increase BCAA catabolism to see if mitochondrial function and cell survival improve and will study the transcriptional controls, such as KLF6, that turn these genes off during injury. Findings could point to new targets for therapies that protect tubule cells in AKI and slow progression to chronic kidney disease.

Who could benefit from this research

Good fit: Ideal candidates for future trials based on this work would be adults experiencing acute kidney injury or people with chronic kidney disease at risk of worsening.

Not a fit: Patients with kidney problems driven primarily by obstruction, infection, or causes unrelated to proximal tubule energy metabolism may not benefit directly from these approaches.

Why it matters

Potential benefit: If successful, this work could identify new ways to protect kidney cells during acute injury and reduce the chance of developing long‑term kidney disease.

How similar studies have performed: Related efforts that boosted fatty‑acid oxidation helped in animal AKI models but have not yet translated to patients, and targeting BCAA catabolism is a newer approach with limited prior clinical testing.

Where this research is happening

Stony Brook, United States

Researchers

About this research

  1. This is an active NIH-funded research project — typically early-stage science, not a clinical trial accepting patient enrollment.
  2. Some NIH-funded labs run parallel clinical studies or seek volunteers for related work. To check, contact the principal investigator or institution listed above.
  3. For full project details, budget, and progress reports, visit the official NIH RePORTER page below.
Last reviewed 2026-06-13 by the Find a Trial editorial team. Information on this page is for educational purposes and is not medical advice. Always consult qualified healthcare professionals about clinical trial participation.