How a protein complex helps genetic messages leave cells in advanced prostate cancer

Function of the METTL3-NUP93 complex-mediated nuclear export of m6A-modified mRNAs in castration-resistant prostate cancer.

NIH-funded research University of Virginia · NIH-11285441

This project looks at whether blocking a protein complex that helps send genetic messages (mRNA) out of the cell nucleus can slow the growth and spread of castration‑resistant prostate cancer.

Quick facts

Grant typeR01 grant
Study typeNIH-funded research
Funding institutionUniversity of Virginia NIH-funded
Lab location1 site (Charlottesville, United States)
Project IDNIH-11285441 on NIH RePORTER

What this research studies

Researchers will examine tumor tissue and laboratory prostate cancer cells to study a protein complex called METTL3‑NUP93 that helps export m6A‑tagged mRNA from the nucleus. They will compare METTL3 and NUP93 levels in normal prostate tissue, primary tumors, and metastatic castration‑resistant prostate cancer using tissue staining. In lab-grown cancer cells they will disrupt the METTL3‑NUP93 interaction with targeted mutations and measure effects on cell growth and movement. Proteomics and molecular methods will track how changes in this complex affect mRNA export and cancer cell behavior.

Who could benefit from this research

Good fit: Men with castration‑resistant or metastatic prostate cancer, especially those willing to provide tumor tissue for research, would be the most relevant candidates for involvement or future trials.

Not a fit: People without prostate cancer or those seeking an immediate therapeutic benefit should not expect direct help from this laboratory‑focused project.

Why it matters

Potential benefit: If successful, this work could reveal a new molecular target for treatments aimed at slowing or stopping castration‑resistant prostate cancer progression.

How similar studies have performed: Previous research has linked METTL3 and nucleoporins to cancer, but studying their interaction in mRNA export specifically in castration‑resistant prostate cancer is relatively novel.

Where this research is happening

Charlottesville, United States

Researchers

About this research

  1. This is an active NIH-funded research project — typically early-stage science, not a clinical trial accepting patient enrollment.
  2. Some NIH-funded labs run parallel clinical studies or seek volunteers for related work. To check, contact the principal investigator or institution listed above.
  3. For full project details, budget, and progress reports, visit the official NIH RePORTER page below.
Conditions Anti-Cancer Agents
Last reviewed 2026-06-13 by the Find a Trial editorial team. Information on this page is for educational purposes and is not medical advice. Always consult qualified healthcare professionals about clinical trial participation.