Finding ways to treat prion diseases by improving how neurons break down proteins.

Restoring neuronal degradative capacity as a therapeutic strategy to treat Prion Disease

NIH-funded research Scripps Research Institute, the · NIH-11040904

This study is looking at prion diseases, which are rare brain conditions caused by bad proteins, to find out how these proteins affect nerve cells and to see if boosting the body's cleanup process can help develop new treatments that might slow down or prevent these diseases.

Quick facts

Grant typeR01 grant
Study typeNIH-funded research
Funding institutionScripps Research Institute, the NIH-funded
Lab location1 site (La Jolla, United States)
Project IDNIH-11040904 on NIH RePORTER

What this research studies

This research focuses on prion diseases, which are rare neurodegenerative conditions caused by misfolded proteins that accumulate in the brain. The study aims to understand how these misfolded proteins affect neuronal function, particularly in axons, and explores the potential of activating autophagy, a cellular process that helps break down and remove these harmful aggregates. By targeting the mechanisms that lead to the accumulation of toxic proteins, the research seeks to develop new therapeutic strategies to restore neuronal health and function. Patients may benefit from novel treatments that could slow down or prevent the progression of these debilitating diseases.

Who could benefit from this research

Good fit: Ideal candidates for this research include individuals diagnosed with prion diseases or Alzheimer's Disease and related dementias.

Not a fit: Patients with other forms of neurodegenerative diseases that do not involve prion proteins may not receive benefit from this research.

Why it matters

Potential benefit: If successful, this research could lead to new therapies that improve the quality of life for patients with prion diseases and related neurodegenerative conditions.

How similar studies have performed: Previous research has shown promise in targeting autophagy to reduce protein aggregates in neurodegenerative models, indicating a potential for success in this approach.

Where this research is happening

La Jolla, United States

Researchers

About this research

  1. This is an active NIH-funded research project — typically early-stage science, not a clinical trial accepting patient enrollment.
  2. Some NIH-funded labs run parallel clinical studies or seek volunteers for related work. To check, contact the principal investigator or institution listed above.
  3. For full project details, budget, and progress reports, visit the official NIH RePORTER page below.
Conditions Alzheimer disease dementiaAlzheimer syndrome
Last reviewed 2026-06-13 by the Find a Trial editorial team. Information on this page is for educational purposes and is not medical advice. Always consult qualified healthcare professionals about clinical trial participation.